B, C and A

All of them

D, A and E

B, D and C

A, E and C

D

A

C

E

B

D

A

C

E

B

Is lowest at the axon hillock since there is the least loss of current at this point  due to summation

Is lowest at the dendrites where current can sum

Is lowest at the axon hillock where there is the highest density of voltage-gated Na⁺ channels

Is lowest at the dendrites since there is the least loss of current at this point 

Is lowest at the dendrites where there is the highest density of voltage-gated Na⁺ channels

Because it is bound to GDP

Because it is bound to PLC-beta 1

Because it is bound to GTP

Because it is bound to Ca⁺⁺

Because it is bound to cyclic AMP

the post-synaptic cell RMP would become hyper-polarized

the post-synaptic cell would activate G proteins

the post-synaptic cell would activate G proteins to activate enzymes

the post-synaptic cell RMP would become de-polarized

the post-synaptic cell RMP would become polarized

Even when the synapse is at the axon hillock the channels activated by the EPSP rapidly become refractory

Most synapses are at the cell body which also receives inhibitory input to cancel the EPSP

Even when the synapse is at the axon hillock the EPSP is too short-lasting an event to allow summation

Most synapses are at the dendrites and there are no voltage-gated Na⁺ channels at the dendrites to produce an AP

Most synapses are at the dendrites and there is too much loss of current from an EPSP by the time it reaches the axon hillock

A neurotransmitter that selectively opens potassium channels will alow potassium to move into the cell

Chloride ions and potassium ions move into the cell to hyperpolarise the cell

A neurotransmitter agent selectively opens ligand-gated chloride channels is the basis for an inhibitory postsynaptic potential

The Nernst potential for chloride is about -70mV, therefore chloride ions tend to move out of the cell along its electrochemical gradient

An increase in the extracellular sodium concentration leads to an inhibitory postsynaptic potential

Alterations in the amount of the depolarization of the pre-synaptic terminal 

Alterations in pre-synaptic facilitation or inhibition

Alterations in the pre-synaptic terminal levels of Ca⁺⁺

Alterations in the size of the depolarization of the pre-synaptic terminal 

Desensitization of the post-synaptic receptors