The vascular smooth muscle will relax, and blood vessel diameter will increase, resulting in organ blood flow being maintained at a normal level

Angiotensin II will be released, causing vasoconstriction to prevent transmission of the increase in arterial pressure to the capillary bed.

This might be caused by a metabolically driven increase in organ blood flow following vascular occlusion

The vascular smooth muscle will contract. The resulting vasoconstriction will maintain blood flow to the organ relatively constant

increase in venous return

increase in cardiac output

decrease in blood volume

increase in heart rate

an increase in acetylcholine release at the sino-atrial node resulting in a decrease in heart rate

an increase in noradrenaline release at the sino-atrial node resulting in a decrease in heart rate

an increase in acetylcholine release at the sino-atrial node resulting in an increase in heart rate

an increase in acetylcholine release at vascular smooth muscle cells to cause vasoconstriction