Why is neurotransmission likely to fail at the neuromuscular junction in individuals with Myasthenia Gravis?

Question

Accepted Answer

there are fewer interactions between ACh and AChR, resulting in smaller end plate potentials

Answer

there is increased binding of ACh and AChR, saturating binding at the end plate

Answer

repeated nerve activation results in the constant production of ACh

Answer

larger end plate potentials are the result of fewer interactions between ACh and AChR

Answer

ACh cannot be released from the nerve terminal due to unsuccessful vesicle docking

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