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In advanced atherosclerotic lesions, what cellular mechanism converts a stable p...

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In advanced atherosclerotic

lesions, what cellular mechanism converts a stable plaque into a vulnerable,

rupture-prone plaque with an expanding necrotic core?

Enhanced

macrophage efferocytosis and rapid resolution of inflammation.

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Upregulation of ABCA1 efflux transporters in vascular smooth muscle cells.

❌

Proliferation of stable collagen fibers driven by statin administration.

❌

Continual macrophage foam cell death (apoptosis/necroptosis) coupled with defective clearance (efferocytosis) of dead cells.

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