Henle's loop and the macula densa in the kidney:

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the macula releases ATP in response to low sodium load (tubulo-glomerular feedback)

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the macula can release renin to increase Na+ reabsorption (via aldosterone)

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the macula is a sensor of GFR, independent of Na+ concentration

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the macula can release ATP that indirectly constricts the afferent arteriole

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Henle's loop generates a gradient in urea concentration in the renal medulla

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the macula is a sensor of Na+ concentration in the lumen, independent of GFR

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Henle's loop terminates into the straight portion of the distal tubule, where the macula densa transduces the activity of NKCC transporter into a chemical signal to the juxta-glomerular apparatus

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Henle's loop expresses the same Na:Cl transporter that is expressed in the whole distal convoluted tubule

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Henle's loop is involved in regulating sodium, potassium and chloride reabsorption

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Henle's loop is involved in regulating sodium reabsorption

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the thick portion of the ascending limb of Henle's loop expresses the same transport systems as the macula densa cells

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Henle's loop and the macula densa in the kidney:

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