It stimulates baroreceptors in the carotid arch to decrease sympathetic outflow.

It inhibits renin production in the kidneys in response to high intrarenal blood pressure.

It increases parasympathetic output to the heart, decreasing cardiac output.

It acts as a potent vasoconstrictor and promotes aldosterone release, leading to sodium retention and increased blood volume.

It blocks AT₁ receptors to cause vasodilation and reduce peripheral vascular resistance.