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Hyperalgesia consists of primary and secondary components.
Primary hyperalgesia is sensitization of bare nerve endings and secondary hyperalgesia is sensitization of encapsulated nerve endings
Primary hyperalgesia is sensitization of the damaged area to non-painful stimuli and secondary hyperalgesia is sensitization of the damaged area to painful stimuli
Primary hyperalgesia is sensitization of the damaged area and secondary hyperalgesia is sensitization of surrounding skin
Primary hyperalgesia is due to sensitization of TRPV1 channels and secondary hyperalgesia due to sensitization of voltage-gated channels
Primary hyperalgesia is sensitization of the damaged area to painful stimuli and secondary hyperalgesia is sensitization of the damaged area to non-painful stimuli
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