They are reactivated in the draining lymph node before leaving to eliminate pathogen

They are able to recognise any peptide from the pathogen, when presented in the "correct" MHC molecule

Some will remain in, and re-circulate through, the lymph nodes to deal with incoming pathogens

The requirement for co-stimulation upon antigen recognition is less than required for naïve T cells

The TH2 cytokine IL-4 causes degranulation of mast cells

TH2 cells cause the cross-linking of B cells necessary for mast cell degranulation

They promote antibody production

They bind the allergen directly to drive inflammation

Not generated in humans

The transfer of B cells specific for the snake venom

A mixture of T and B cells specific for the snake venom

First generated in you as a vaccination

The affinity of the IgM and IgG will always be the same, even if the level of each antibody differs

The affinity of IgG is maximal after the primary response and decreases upon further immunisations

The order and the amount of IgM and IgG produced will be the same at each time point

The secondary responses will predominantly be associated with IgG

They do not contain protein

Polysaccharides do not induce antibody responses

Polysaccharides are toxic to T cells

T cells are not able to present polysaccharides to B cells

It will leave the skin, enter the blood circulation, and migrate to the spleen to mount an immune response

It will express more MHC molecules to increase the presentation of pathogen peptides

It will proliferate, and then secrete cytokines and chemokines to attract naïve T cells to the site of infection

It will proliferate to clonally expand so more pathogen peptides can be presented to more T cells

It is found on all innate cells as the universal Fc receptor to promote phagocytosis

It enables IgG to have a half-life of 3 months

It enables maternal IgG to be transferred to the foetus

It enables IgG to be expressed in maternal breast milk

Location of activation

MHC class

Strength of the TCR binding

Cytokine

That the microbe is rapidly phagocytosed by the B cell

The microbe is able to engage multiple BCRs at the same time

The spontaneous activation of B cells in the hope of a useful response- similar to the alternative complement pathway

That it must first invade/infect the B cell to activate it from the cytosol.