At the post-ganglionic synapse

In the CNS

At the ganglion

At the pre-ganglionic synapse

In the pre-synaptic terminals

Produce fast non-genomic actions of lipid soluble hormones

To make sure that no one gets drunk or mis-behaves at formals

Activate 2^nd messengers

To cause the release of Ca⁺⁺ from intra-cellular stores

Hold nuclear receptors for steroid hormones in place for binding with the hormone

Beta adrenaline

Only nicotinic ACh

ACh or NA

Only muscarinic ACh

Only ACh

Have a single binding domain for the G protein, another for the hormone and a third for the DNA

Have separate binding domains for the hormone and for the DNA

Are capable of activating the enzymes that activate 2nd messengers after binding the hormone and the chaperone protein

Are G protein coupled receptors with a binding site for the hormone and one for the chaperone protein

Are enzymes that activate 2nd messengers after binding the hormone and the chaperone protein

They are changes in the resting membrane potential rather than events independent of the RMP

They both involve transmembrane ionic movements through voltage-gated ion channels

They both require a threshold to be reached before they occur

They both require the influx of Ca⁺⁺ into the cell through voltage-gated Ca⁺⁺ channels

They both are depolarizations of the resting membrane potential

modifying the integration of EPSP's and IPSP's at the pre-synaptic axon hillock.

modifying the release of transmitter from an excitatory pre-synaptic neuron.

modifying the permeability of the post-synaptic neuron only to Na⁺.

modifying the ability of the post-synaptic neuron to respond to any of its inputs.

producing a smaller EPSP in the post-synaptic neuron.

Increased release of neurotrnasmitter through pre-synaptic GPCR effects

Changes in neuronal post-synaptic adaptation over hours to days

The effect of pre-synaptic facilitation

Increased release of neurotrnasmitter through pre-synaptic 2^nd messenger effects

Changes in synaptic information transfer efficiency over hours to days

only by binding to membrane-bound receptors that form ion channels.

that are always a depolarization of the post-synaptic resting membrane potential.

only by binding to receptors that are protein complexes made up of five sub-units.

that may be a depolarization or a hyper-polarization of the post-synaptic RMP.

through receptors that selectively allow the movement of only one ion.

IPSPs take the membrane potential further from threshold

An IPSP is considered an inhibitory response

An IPSP is a hyperpolarisation

IPSPs increase the likelihood of a post-synaptic AP

IPSPs decrease the likelihood of a post-synaptic AP