They both modulate the docking proteins so that vesicles fusion in changed, even if in different ways

They both modulate the priming and docking processes for vesicles fusion, even if in different ways

They selectively modulate only specific inputs to target cells

They both modulate how neurotransmitter is produced and stored in the pre-synaptic terminal 

They modulate pre-synaptic terminal voltage-gated Na⁺⁺ levels

 G-protein activation in the presynaptic terminal

The is a large efflux of Ca++ from the presynaptic terminal

Voltage gated K+ ion channels are closed

Ligand-gated channels open to allow Na+ entry into the postsynaptic neuron

Synaptic vesicles bind to the postsynaptic membrane

Allow selective inhibition of only some inputs to a target neuron

Are forms of hard-wired synaptic plasticity

Illustrate the way in which modulation of pre-synaptic Ca⁺⁺ alters post-synaptic neural responses

Illustrate that synaptic signalling is subject to the influence of past activity

Allow selective activation of only some inputs to a target neuron

It allows for temporal but not spatial summation of post-synaptic potentials

It allows for a high degree of selectivity in what inputs the target neuron will respond to

It would produce a larger post synaptic response to all inputs

It would produce a smaller post synaptic response to all inputs

It allows for summation of post-synaptic potentials

Down regulation of receptor production

Sequestering receptors intracellularly

Altering the resting membrane potential of the cell

Binding of chemicals to alter receptor structure

Inhibition of vesicle fusion

The cholinergic AP

The EPP

The APP

The IPSP

The EPSP

Muscarine receptors

GPCR

Nicotine adrenergic receptors

MUscarinic cholinergic receptors

Nicotine cholinergic receptors

The brain

The ganglion

The CNS

The ANS

The spinal cord

D

A

C

E

B

Causing the release of Ca⁺⁺

Dissociating into three parts

Dissociating into two parts

Binding to GTP

Causes the depolarization of the target cell