ruptured ventricle or aorta.

congestive heart failure.

cardiac arrhythmias and fibrillation.

cerebrovascular accident.

Decreased renal excretory function

Increased activity of the renin- angiotensin - aldosterone system

Increased sensitivity of the kidneys to the action of vasopressin

Decreased secretion of components of the kallikrein-kinin system and prostaglandins

repeated vasospasms.

accumulated lipids, cells, and fibrin where endothelial injury has occurred.

a torn arterial wall and blood clots.

thrombus forming on damaged walls of veins.

a state, when the heart cannot cope with increased workload

a state, when myocardial contractility initially decreases

increased frequency and strength of myocardial contractions

increased heart size

destruction of ß-cells of the islets of Langerhans

damage to the exocrine part of the pancreas

blocking of insulin by autoantibodies

decreased density of insulin receptors

activation of the systemic inflammatory response

glycation of macromolecules of blood and components of the vascular wall

exhaustion of the body

considerable shifts of physicochemical parameters of extracellular fluid

formation of IgE antibodies

formation of sensitized T-lymphocytes

implementation of antibody-dependent cytotoxicity

action of histamine, serotonin

increases

decreases

does not change

spherical transformation of platelets, formation of thromboxane A2 and release of proaggregants/procoagulants from granules

sticking of platelets and erythrocytes due to electrostatic attraction

precipitation of blood plasma proteins on the platelet membrane, their coagulation and formation of active prothrombinase

receptor-mediated attachment of platelets to the vascular wall with the formation of a monolayer

hypoosmolar hypohydration

isosmolar hyperhydration

isosmolar hypohydration

hyperosmolar hypohydration