Atropine will block acetylcholine binding to receptors on the post synaptic muscle fiber and the force of muscle contraction will be reduced

 Atropine will block acetylcholine binding to receptors on the post synaptic muscle fiber an muscle contraction will be inhibited

Atropine will have no effect on neuromuscular transmission

Acetylcholine will hang around longer in the synaptic cleft

An increase in the number of Na+ channels opening in the post synaptic muscle fiber and an increase in the end plate potential

The motor neuron terminal has vesicles each with a large amount of neurotransmitter.

The structure of the NMJ maximises the opportunity for ACh to remain in the cleft.

The folded surface of the motor end plate provides a large surface area that allows for a high density of ACh receptors

Presynaptic facilitation increases the amount of transmitter released and the size of the postsynaptic response

ACh binds to its receptors for longer than at most other receptor-transmitter interactions

voltage gated channels

chemically gated channels

diffusion gated channels

passive channels

mechanically gated channels

D and E

A

A, B, D and E

A and C

B and D

Sarin gas

Organophosphorus pesticides

Lexapro

Curare

Physostigmine

B, C and A

All of them

D, A and E

B, D and C

A, E and C