Moving docking proteins away from the active zone

Putting Ca⁺⁺ into internal stores to make it unavailable

Increasing the efflux of K⁺ from the target cell

Using pre-synaptic inhibition

Using enzymes to destroy the transmitter in the cleft

synthesis, storage, exocytosis, reuptake

exocytosis, postsynaptic binding, synthesis, reuptake

storage, deactivation, synthesis, exocytosis

deactivation, postsynaptic binding, synthesis, storage

reuptake, synthesis, deactivation, exocytosis

Regulated synthesis machinery for transmitter to be found in the cell body

High affinity post-synaptic receptors and a mechanism for termination

A mechanism in the synaptic cleft for termination of action of the chemical

Regulated release, binding to post-synaptic receptors and a mechanism for termination

BNinding to post-synaptic receptors leading to depolarisation

Uptake into glial cells

Diffusion out of the synapse

Uptake into the post-synaptic neuron

Reuptake into the neuron

Degradation by enzymes

A region which is only found on diffuse type synapses where transmitter release occurs

The area on the post synaptic cell where receptors are concentrated

An unmyelinated region on the neuronal axon where release of neurotransmitter occurs

The area of a neuron which receives inputs from other neurons

The binding site of ionotropic receptors to allow influx of ions

To overcome the longer synaptic delay at peptidergic synapses

To overcome pre-synaptic facilitation

To accumulate enough Ca⁺⁺ in the terminal for more transmitter release

To activate enough ATP in the terminal for more transmitter release

To overcome pre-synaptic inhibiiton

Ca2+ entry into the axon terminal would no longer cause a concentrated release of neurotransmitter

 Neurotransmitter would continuously leak out of the cell

There would be a more concentrated release of neurotransmitter

There would be a decreased chance of neurotransmitter eliciting a response in the post synaptic cell

More neurotransmitter would be degraded by cellular enzymes

The influx of Ca++ into the presynaptic terminal

Activation of ligand-gated ion Ca++ channels in the active zone

The opening of ligand-gated ion Ca++ channels

The production of acetylcholinesterase

The hyperpolarisation of the synaptic terminal