Hypo-osmotic and Hypo-tonic, and will cause lysis of RBCs

Hyper-osmotic and Hypo-tonic, and will cause lysis of RBCs

Iso-osmotic and Iso-tonic, and will have no effect on RBC volume

Iso-osmotic and Hypo-tonic, and will cause lysis of RBCs

Hyper-osmotic and Hypo-tonic, and will cause crenation of RBCs

Hyper-osmotic and Iso-tonic, and will have no effect on RBC volume

Hyper-osmotic and Hypo-tonic, and will cause lysis of RBCs

Hyper-osmotic and Iso-tonic, and will cause crenation of RBCs

Hyper-osmotic and Iso-tonic, and will cause lysis of RBCs

Iso-osmotic and Hypo-tonic, and will cause lysis of RBCs

One group of muscle fibres

One group of motor units

One skeletal muscle fibre

One muscle fibre

One muscle group

Is a muscarinic ACh receptor that allows the influx of Na⁺ and Ca⁺⁺ at the motor end plate

Is a muscarinic ACh receptor that allows the influx of Na⁺ and efflux of K⁺ at the motor end plate

Is a nictonic ACh receptor that allows influx of Ca⁺⁺

Is a nictonic ACh receptor that allows the influx of Na⁺ and efflux of K⁺ at the motor end plate

Is a muscarininc ACh receptor that allows the influx of Na⁺ of K⁺ at the motor end plate

The termination of muscle contraction doesn't occur

Neurotransmitter wont be released to the post synaptic muscle cell

 No effect because neuromuscular transmission does not rely on acetylcholinesterase

Muscle fibres of the post synaptic muscle cell cannot contract

Acteylcholine cant bind with the receptors in the neuromuscular end plate

there are fewer interactions between ACh and AChR, resulting in smaller end plate potentials

there is increased binding of ACh and AChR, saturating binding at the end plate

repeated nerve activation results in the constant production of ACh

 larger end plate potentials are the result of fewer interactions between ACh and AChR

ACh cannot be released from the nerve terminal due to unsuccessful vesicle docking