Decreased cardiac output.

Decreased stroke volume.

Increased pulmonary artery pressure.

Decreased heart rate.

Increased peripheral vascular resistance.

Fibular artery.

Great saphenous vein.

Posterior tibial artery.

Small saphenous vein.

Anterior tibial artery.

Large nerve bundles with multipolar neurons passing through the subendothelial layer of the endocardium.

A thick layer of adipose tissue in the endocardium.

A thin layer of mesothelium forming the innermost layer of the endocardium.

Irregular folding of the luminal surface due to the pectinate muscle.

A high density of capillaries in the myocardium.

Carotid sinus baroreceptor activity increases.

Venous return increases.

Heart rate falls.

Peripheral resistance decreases.

Mean arterial pressure falls transiently (briefly).

ACEi drugs have direct inotropic actions on the heart.

ACEi drugs may assist with controlling oedema by increasing the effects of aldosterone.

ACEi drugs work by blocking the vascular and renal angiotensin II receptors, reducing its actions.

ACEi drugs may promote cardiac hypertrophy of the cardiac muscle, which is beneficial in heart failure.

ACEi drugs address the compensatory mechanisms that are activated in heart failure.

25mm/min

25mm/sec

25cm/sec

2.5mm/sec

By a pseudonym

By their first name only

By the group number

By their full name

By their student number

It is in the anterior axillary line

It is in the midclavicular line

It is on the right side of the chest.

It is in the posterior axillary line

It is in the midaxillary line

Left atrium

Left arm

Left leg